Triple
T19992814
| Position | Surface form | Disambiguated ID | Type / Status |
|---|---|---|---|
| Subject | NMDA receptor |
E494104
|
entity |
| Predicate | encodedBy |
P14248
|
FINISHED |
| Object | GRIN3B |
—
|
NE NERFINISHED |
How this triple was built (3 steps)
Every LLM step that produced this triple, in pipeline order — named-entity classification, the disambiguation choices (the exact options shown, with the pick highlighted), and the generated description. The batch + timestamp of each is in the Provenance table below.
NER
Named-entity recognition
gpt-5-mini
Instruction
Given a phrase, classify it is english named entity (e.g., persons, organizations, works of art) in Latin script, or not (e.g., literals, dates, URLs, verbose phrases). For disambiguation, the statement where the phrase occurs as object is also given. Please return a JSON object with `phrase` (string, the phrase being analyzed) and `is_ne` (boolean, indicating whether the phrase is a Named Entity).
Input
Phrase: GRIN3B | Statement: [NMDA receptor, encodedBy, GRIN3B]
NED1
Entity disambiguation (via context triple)
gpt-5-mini-2025-08-07
Target entity: GRIN3B Context triple: [NMDA receptor, encodedBy, GRIN3B]
-
A.
GRIN3A
GRIN3A is a gene that encodes a subunit of the N-methyl-D-aspartate (NMDA) receptor, a key glutamate-gated ion channel involved in synaptic transmission and plasticity in the central nervous system.
-
B.
GRIN1
GRIN1 is a gene that encodes an essential subunit of NMDA-type glutamate receptors, which play a key role in synaptic transmission and plasticity in the central nervous system.
-
C.
GRIN2B
GRIN2B is a gene that encodes the GluN2B subunit of NMDA-type glutamate receptors, which are crucial for synaptic plasticity, learning, and memory in the brain.
-
D.
GRIN2A
GRIN2A is a gene that encodes the GluN2A subunit of NMDA-type glutamate receptors, which are critical for synaptic plasticity, learning, and memory in the brain.
-
E.
GNAS
GNAS is the abbreviation for the Georgian National Academy of Sciences, the leading scientific institution in Georgia that coordinates and promotes research across various disciplines.
- F. None of above. chosen
- G. Unsure - the case is ambiguous/there is not enough information to decide.
NED2
Entity disambiguation (via description)
gpt-5-mini-2025-08-07
Target entity: GRIN3B Target entity description: GRIN3B is a human gene that encodes the GluN3B subunit of NMDA-type glutamate receptors involved in excitatory neurotransmission and synaptic plasticity in the central nervous system.
-
A.
GRIN3A
GRIN3A is a gene that encodes a subunit of the N-methyl-D-aspartate (NMDA) receptor, a key glutamate-gated ion channel involved in synaptic transmission and plasticity in the central nervous system.
-
B.
GRIN1
GRIN1 is a gene that encodes an essential subunit of NMDA-type glutamate receptors, which play a key role in synaptic transmission and plasticity in the central nervous system.
-
C.
GRIN2B
GRIN2B is a gene that encodes the GluN2B subunit of NMDA-type glutamate receptors, which are crucial for synaptic plasticity, learning, and memory in the brain.
-
D.
GRIN2A
GRIN2A is a gene that encodes the GluN2A subunit of NMDA-type glutamate receptors, which are critical for synaptic plasticity, learning, and memory in the brain.
-
E.
GNAS
GNAS is the abbreviation for the Georgian National Academy of Sciences, the leading scientific institution in Georgia that coordinates and promotes research across various disciplines.
- F. None of above. chosen
Provenance (2 batches)
The batch behind each pipeline step, in order, with when it ran. Timestamps are batch-level — stages were processed in waves, so the object chain (NER → NED1 → NEDg → NED2) reads in order, but predicate / elicitation batches can sit in a different wave.
| Step | Stage | Batch ID | Status | When |
|---|---|---|---|---|
| creating | Elicitation | batch_69da626a67648190af9653832a3aeced |
completed | April 11, 2026, 3:02 p.m. |
| NER | Named-entity recognition | batch_69e65fe2036c8190b9f313215ad44e87 |
completed | April 20, 2026, 5:18 p.m. |
Created at: April 11, 2026, 3:31 p.m.