Triple
T19992803
| Position | Surface form | Disambiguated ID | Type / Status |
|---|---|---|---|
| Subject | NMDA receptor |
E494104
|
entity |
| Predicate | hasSubunit |
P747
|
FINISHED |
| Object | GluN2B |
—
|
NE NERFINISHED |
How this triple was built (3 steps)
Every LLM step that produced this triple, in pipeline order — named-entity classification, the disambiguation choices (the exact options shown, with the pick highlighted), and the generated description. The batch + timestamp of each is in the Provenance table below.
NER
Named-entity recognition
gpt-5-mini
Instruction
Given a phrase, classify it is english named entity (e.g., persons, organizations, works of art) in Latin script, or not (e.g., literals, dates, URLs, verbose phrases). For disambiguation, the statement where the phrase occurs as object is also given. Please return a JSON object with `phrase` (string, the phrase being analyzed) and `is_ne` (boolean, indicating whether the phrase is a Named Entity).
Input
Phrase: GluN2B | Statement: [NMDA receptor, hasSubunit, GluN2B]
NED1
Entity disambiguation (via context triple)
gpt-5-mini-2025-08-07
Target entity: GluN2B Context triple: [NMDA receptor, hasSubunit, GluN2B]
-
A.
GluN2A
GluN2A is a subunit of NMDA-type glutamate receptors that plays a key role in synaptic transmission and plasticity in the central nervous system.
-
B.
NMDA receptor
The NMDA receptor is a glutamate-activated ion channel in the brain that plays a key role in synaptic plasticity, learning, and memory, and is implicated in various neurological and psychiatric disorders.
-
C.
AMPA receptors
AMPA receptors are fast-acting ionotropic glutamate receptors in the brain that mediate most rapid excitatory synaptic transmission and play key roles in synaptic plasticity and learning.
-
D.
GABA-A receptor
The GABA-A receptor is a ligand-gated chloride ion channel in the central nervous system that mediates fast inhibitory neurotransmission and is a primary target of many sedative, anxiolytic, and hypnotic drugs.
-
E.
GRIN2A
GRIN2A is a gene that encodes the GluN2A subunit of NMDA-type glutamate receptors, which are critical for synaptic plasticity, learning, and memory in the brain.
- F. None of above. chosen
- G. Unsure - the case is ambiguous/there is not enough information to decide.
NED2
Entity disambiguation (via description)
gpt-5-mini-2025-08-07
Target entity: GluN2B Target entity description: GluN2B is a subunit of NMDA-type glutamate receptors that plays a key role in synaptic plasticity, learning, and neurodevelopment in the central nervous system.
-
A.
GluN2A
GluN2A is a subunit of NMDA-type glutamate receptors that plays a key role in synaptic transmission and plasticity in the central nervous system.
-
B.
NMDA receptor
The NMDA receptor is a glutamate-activated ion channel in the brain that plays a key role in synaptic plasticity, learning, and memory, and is implicated in various neurological and psychiatric disorders.
-
C.
AMPA receptors
AMPA receptors are fast-acting ionotropic glutamate receptors in the brain that mediate most rapid excitatory synaptic transmission and play key roles in synaptic plasticity and learning.
-
D.
GABA-A receptor
The GABA-A receptor is a ligand-gated chloride ion channel in the central nervous system that mediates fast inhibitory neurotransmission and is a primary target of many sedative, anxiolytic, and hypnotic drugs.
-
E.
GRIN2A
GRIN2A is a gene that encodes the GluN2A subunit of NMDA-type glutamate receptors, which are critical for synaptic plasticity, learning, and memory in the brain.
- F. None of above. chosen
Provenance (2 batches)
The batch behind each pipeline step, in order, with when it ran. Timestamps are batch-level — stages were processed in waves, so the object chain (NER → NED1 → NEDg → NED2) reads in order, but predicate / elicitation batches can sit in a different wave.
| Step | Stage | Batch ID | Status | When |
|---|---|---|---|---|
| creating | Elicitation | batch_69da626a67648190af9653832a3aeced |
completed | April 11, 2026, 3:02 p.m. |
| NER | Named-entity recognition | batch_69e65fe2036c8190b9f313215ad44e87 |
completed | April 20, 2026, 5:18 p.m. |
Created at: April 11, 2026, 3:31 p.m.